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Based upon "Resonant Excitation Of Sexual Orgasms - Tao Of Love Coupling"
by Newman K. Lin, Ph.D., PE, a bridge between the Eastern Taoism Sexuality and the Western Engineering Science.==> [ORDERING THE BOOK]< =>[Why?]
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Warning: This is NOT an XXX Website, But we deal with Multiple, Sexual Orgasms and Impotence!
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Case Title:  Hysterectomy killed her clitoris for no sexual orgasm.
Reader : 4/29/2004>
i am a 49 year old nurse that had my uterus removed 10 years ago and since the surgery i have very little feeling in my clitoris, compared to before the surgery, when i could climax with very little effort. now it take so much effort. i've tried testosterone creams and esstest with very little results. do you have any sugestions? thank you,
Dr. Lin: 4/29/2004>  
 I call this is a death of the clitoris after mechanically cutting, chemically blocking, or mechanically/chemically damaging the L1 and L2 sympathetic sensory/motoring nervous circuits in clitoris, G-spot and uterus/cervix. The L1 and L2 pathways are linked to the hypothalamus/pituitary dopamine nervous system for a release of oxytocin. The neurotransmitter acetylcholine is responsible for the sensory and sympathetic preganglionic nervous transduction; after the sympathetic preganglionic axion, norepinephrine takes over the transduction of the nervous pulses into the sympathetic postganglionic nerves to the effector for orgasmic contraction which is synchronized by the flooding of norepinephrine and adrenalin to the tissue and smooth muscle via in the blood circulation.
Acetylcholine and its secondary neurotransmitters NO (Nitric Oxide) are also responsible for the parasympathetic nervous dilation of the arteries and thus the erection of the clitoris and G-spot, while norepinephrine and adrenalin, when bounded into the beta receptors, are responsible for the sympathetic nervous dilation of the arteries and the erection of the g-spot and clitoris.
Without the erection of the clitoris and G-spot, the androgen hormone and the liver 5-alpha reductase won't reach the clitoris and G-spot testosterone and DHT receptors. Then, the clitoris and G-spot gradually shrink and then eventually die.
Your androgen hormone replacement therapy can not stimulate your hypothalamus and pituitary to enhance the potential production of oxytocin and to suppress the prolactin production. The problem is: the oxytocin release requires the L1 and L2 sensory nervous electric pulses (action potential ) acting on the hypothalamus and pituitary's dopamine nervous receptors.
You have to re-establish the partial L1/L2 nervous transponding and  to revive the clitoris and G-spot if you want to achieve orgasm again.
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